Long COVID continues to debilitate a significant number of U.S. adults — 7.5%, or 1 in 13,1 are struggling with a range of symptoms that make up this complex condition. Among those who have had COVID-19, 11% say they currently have long COVID,2 which often includes unrelenting fatigue, respiratory symptoms, neurological difficulties and joint or muscle pain, all of which may become worse after physical or mental exertion.3
Long COVID symptoms share many similarities with post-jab injuries, and it’s likely both are rooted in mitochondrial dysfunction. Now, however, there’s a growing trend to label long COVID and injuries from COVID-19 shots “functional neurological disorders” (FND), making some patients feel like the medical community thinks their symptoms are “all in their head.”
In an article for TNR, journalist Natalie Shure writes, “The most direct precursor to FND is something you’ve probably heard of: hysteria.”
The Front Line COVID-19 Critical Care Working Group’s (FLCCC) I-RECOVER20 protocol can be downloaded in full,21 giving you step-by-step instructions on how to treat long COVID22 and/or reactions from COVID-19 injections.23 I also recently summarized strategies to optimize mitochondrial health if you’re suffering from long COVID, with a focus on boosting mitochondrial health.
For starters, to allow your body to heal you’ll want to minimize EMF exposure as much as possible. Your diet also matters, as the cristae of the inner membrane of the mitochondria contains a fat called cardiolipin, the function of which is dependent on the type of fat you get from your diet.
The type of dietary fat that promotes healthy cardiolipin is omega-3 fat, and the type that destroys it is omega-6, especially linoleic acid (LA), which is highly susceptible to oxidation. So, to optimize your mitochondrial function, you want to avoid LA as much as possible, and increase your intake of omega-3s.
Primary sources of LA include seed oils used in cooking, processed foods and restaurant foods made with seed oils, condiments, seeds and nuts, most olive oils and avocado oils (due to the high prevalence of adulteration with cheaper seed oils), and animal foods raised on grains such as conventional chicken and pork.
Another major culprit that destroys mitochondrial function is excess iron — and almost everyone has too much iron. Copper is also important for energy metabolism, detoxification and mitochondrial function, and copper deficiency is common. Other strategies include sun exposure and near-infrared light therapy, time-restricted eating, NAD+ optimizers and methylene blue, which can be a valuable rescue remedy.
Whether long COVID has a functional element to it or not, each individual suffering deserves access to the full range of potential treatments. Unfortunately, this often isn’t the case, especially if symptoms are dismissed as purely psychological in nature. If you improve your mitochondrial function and restore the energy supply to your cells, you’ll significantly increase your odds of reversing the problems caused by the jab or the virus.
One case involves Maddie de Garay, who was a healthy 12-year-old when she signed up for Pfizer’s COVID-19 trial for 12- to 15-year-olds. She suffered a severe systemic adverse reaction to her second dose of the shot, however, and struggled through 11 ER visits and four hospital admissions in the year and a half that followed.
Injuries from the shot have left her unable to walk or eat — she receives her nutrition via a feeding tube — and suffering from constant pain, vision problems, tinnitus, allergic reactions and lack of neck control.16
As though the physical trauma wasn’t enough, Maddie and her family were continually dismissed by the medical professionals put in place to help, ignored by the U.S. Food and Drug Administration and denied the care needed to help Maddie.
In Pfizer’s April 2021 disclosure of Maddie’s case to the FDA, it’s stated only that she had “functional abdominal pain.”17 Then, a day before Pfizer submitted their request for emergency approval of the COVID-19 shot for 12- to 15-year-olds to the FDA, they added functional neurological disorder as a diagnosis in Maddie’s chart,18 blaming the side effects from the shot on FND.
Further, once this assessment was made, her physician, Dr. Amal Assa’ad at Cincinnati Children’s Hospital, went so far as to advise against any further investigation, even though Maddie was a participant in a clinical trial:19
“My assessment is that Madeline has a functional impairment that is not organic in nature … I also discourage further work up since this is usually detrimental in functional disorders because it drives the patient to thinking that there must be something wrong that is indicating all this work up. It also delays the necessary psychologic intervention that is needed to help resolve the functional disorder.”
In an article for TNR, journalist Natalie Shure writes, “The most direct precursor to FND is something you’ve probably heard of: hysteria.”4 For centuries, women were diagnosed with “hysteria” to describe a mental disorder that could give rise to physical and other symptoms ranging from seizures and anxiety to pain and paralysis.
It was, in short, a catch-all diagnosis used to categorize symptoms that weren’t otherwise understood or solvable using the mainstream medical treatments of the time. Eventually, much controversy and research suggested it was the hysteria diagnosis that was the delusion.
The medical community was then left to go back where it started from — a range of troubling symptoms, such as myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS), with no obvious solution still existed. “In the 1990s and early 2000s, it was becoming clear that illnesses previously known as hysteria hadn’t simply vanished,” Shure wrote:5
“[Researchers, including neuropsychiatrist Alan Carson,] began to study the symptoms with a neurological lens, conceiving of them as misfiring brain signals rather than a Freudian cry for help.
Rebranding hysteria as FND was to reject the notion that the best way to understand functional paralysis was as a subconscious repression of childhood memories. Rather, it was an interruption in the brain processing that facilitates the executive function of your legs — a blip that could be triggered by all sorts of things.”
It’s now being suggested that “some post-COVID symptoms may be produced by the brain,” Shure notes. “Does that make them any less real?” For instance, half of people with long COVID symptoms fit the criteria to be diagnosed with ME/CFS and some in the community have suggested the symptoms could be due to ME/CFS, which often flares up after viral infection.6
But it’s far from that simple, as symptoms of long COVID include everything from shortness of breath and pounding heart to dizziness, brain fog and depression. Even the CDC states:7
“People with post-COVID conditions may develop or continue to have symptoms that are hard to explain and manage. Clinical evaluations and results of routine blood tests, chest x-rays, and electrocardiograms may be normal.
The symptoms are similar to those reported by people with ME/CFS (myalgic encephalomyelitis/chronic fatigue syndrome) and other poorly understood chronic illnesses that may occur after other infections. People with these unexplained symptoms may be misunderstood by their healthcare providers, which can result in a long time for them to get a diagnosis and receive appropriate care or treatment.”