Just One Dose of Vitamin D Can Prevent The Progression of Multiple Sclerosis – 9 Day Remission In 92 Percent of Subjects

One single dose of calcitriol, the metabolically active version of vitamin D, followed by ongoing vitamin D supplementation has been shown to prevent the progression of multiple sclerosis, according to new research.

Calcitriol (vitamin D3), is transformed in the liver and kidneys into 25- hydroxyvitamin D (25(OH)D), the non-active ‘storage’ form, and 1,25-dihydroxyvitamin D (1,25(OH)2D), the biologically active form that is tightly controlled by the body.
Several studies have reported that the D3 form of the vitamin is more potent that D2, with a study led by Robert Heaney, MD, from Creighton University in Nebraska reporting earlier last year that D3 was 87% more potent than D2 (Journal of Clinical Endocrinology & Metabolism, doi: 10.1210/jc.2010-2230).
Vitamin D3 has been show to protect us from background radiation, cancerheart failure, and many other diseases by influencing hundreds of genes.

While our bodies do manufacture vitamin D on exposure to sunshine (UV-B radiation with a wavelength between 290 and 315 nm), the levels in some northern countries are so weak during the winter months that our body makes no vitamin D at all.

Halts Multiple Sclerosis 

Writing in the Journal of Neuroimmunology. the US-based team investigated the potential of different combinations of vitamin D therapy had benefit on the course of multiple sclerosis (MS) in a mouse model of the disease known as experimental autoimmune encephalomyelitis (EAE).
Led by Professor Colleen Hayes from the University of Wisconsin-Madison, the team assessed the outcomes of varying combinations of the active vitamin D hormone calcitriol and vitamin D3 from dietary supplements — finding that an initial single oral dose of calcitriol followed by daily supplementation with dietary vitamin D3 “was a runaway success.”
“One calcitriol dose plus vitamin D3 supplementation sustainably reversed clinical EAE signs without inducing hypercalcemia,” explained the research team – noting that the protocol ‘rapidly and transiently’ increased T regulatory (Treg) immune cells in the central nervous system of the mice and sustainably reduced CNS CD4+ T cells, and spinal cord and optic nerve pathology, “thereby promoting clinical recovery.”
“All of the animals just got better and better, and the longer we watched them, the more neurological function they regained,” explained Hayes.
The Tap Blog is a collective of like-minded researchers and writers who’ve joined forces to distribute information and voice opinions avoided by the world’s media.

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