New Insight: Alzheimer’s Disease Linked to Pineal Gland Calcification from Fluoride
September 30th, 2014
Big Pharma Fails to Treat Alzheimer’s
- In 2011, Bristol-Myers Squibb tried to develop a gamma-secretase inhibitor (BMS-708163. Test results were less than promising.
- In 2012, Pfizer announced that it was abandoning further development of intravenous formulations of bapineuzumab, an anti-amyloid antibody, after the failure of its Phase II study.
- In 2013, Baxter International reported that its immune-bolstering treatment had ended in a failure after trying to create a drug called Gammagard out of components of the human immune system. It also failed at removing amyloid plaques from the brain.
Research Reveals Link Between Calcification of the Brain and Alzheimer’s Disease
“Fluoride is likely to cause decreased melatonin production and to have other effects on normal pineal function, which in turn could contribute to a variety of effects in humans.” (National Research Council 2006).
“Fluoride does not accumulate in brain. Of all tissues, brain has the lowest fluoride concentration [Jenkins, 1991; Whitford, 1996; Ekstrand, 1996]. It is generally agreed that the blood-brain barrier restricts the passage of fluoride into the central nervous system. The human pineal gland is outside the blood-brain barrier [Arendt, 1995].It is one of a few unique regions in the brain (all midline structures bordering the third and fourth ventricles) where the blood-brain barrier is weak. Cells in these regions require direct and unimpeded contact with blood[Rapoport, 1976]. Therefore, pinealocytes have free access to fluoride in the bloodstream. This fact, coupled with the presence of HA, suggest that the pineal gland may sequester fluoride from the bloodstream.”